First, confirm the heart rate. In bradycardia, it is often < 50 bpm. However, any rate that causes symptoms needs prompt attention, even above 50.
- Support the airway and breathing. In addition, give supplemental oxygen if the patient is hypoxic.
- Attach a cardiac monitor right away. Also record blood pressure and pulse oximetry.
- Establish IV access. Then obtain a 12-lead ECG if it is available. Do not delay treatment to get it.
Think early: Hypoxia, drug toxicity, ischaemia, and electrolyte problems can all cause bradycardia. Therefore, look for these triggers from the start.
For the full official protocol, see the
2020 AHA Guidelines for CPR and ECC
(external link — opens in new tab).
Treating the root cause is just as important as treating the slow heart rate itself. Therefore, look for and fix any triggers while you prepare for further care.
- First, make sure the airway is open. Assist ventilation if needed.
- Next, review current medications and any possible toxins. For example, calcium-channel blockers, beta-blockers, and digoxin are common culprits.
- Finally, consider myocardial ischaemia, hypoxia, and electrolyte problems such as high potassium (hyperkalemia).
For more on drug-induced bradycardia, see this
peer-reviewed PubMed review.
Not all slow heart rates need treatment. However, when bradycardia causes any of the following signs, it becomes a medical emergency.
- Low blood pressure (hypotension)
- Sudden confusion or altered mental status
- Signs of shock, such as pale, cold, or clammy skin
- Chest pain that suggests ischaemia
- Acute heart failure or pulmonary oedema
If none of these signs are present, monitor the patient closely and observe for any changes.
If any sign is present, move immediately to the treatment steps below.
Once you confirm symptomatic bradycardia, start treatment without delay. First, try atropine. If atropine does not work, move to the alternatives below.
Atropine (IV) — First Choice
- Give the first dose of 1 mg IV as a bolus.
- Repeat every 3–5 minutes as needed.
- Stop when you reach the maximum total dose of 3 mg.
Atropine works by blocking vagal tone. Therefore, it is most effective in sinus bradycardia or AV block at the nodal level.
If Atropine Is Not Effective
- Start transcutaneous pacing right away, and/or
- Dopamine infusion: typical range 5–20 mcg/kg/min. Titrate to the patient’s response.
- Epinephrine infusion: typical range 2–10 mcg/min. Similarly, titrate to response.
Choose infusions and pacing according to local protocols, patient physiology, and expert guidance. In addition, reassess frequently after each intervention.
When atropine and infusions are not enough, pacing is the next step. There are two main options to consider.
- Start transcutaneous pacing (TCP) for any unstable patient who has not responded to atropine. It is non-invasive and fast to set up.
- Consider transvenous pacing if instability continues. In addition, get expert consultation early whenever the situation is unclear or worsening.
Learn more about pacing techniques in this
American Heart Association overview of pacemakers.
Common Reversible Causes
Always search for a reversible cause. Treating the cause often resolves the bradycardia on its own. Below are the three main groups to check.
Cardiac
- Myocardial ischaemia or infarction
- Conduction system disease
Drugs & Toxins
- Calcium-channel blockers
- Beta-blockers
- Digoxin toxicity
Metabolic & Other
- Hypoxia
- Hyperkalemia (high potassium)
- Hypothermia
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Bottom Line
The adult bradycardia algorithm has a clear order. First, stabilise the patient. Next, find the cause. Then treat any symptomatic bradycardia with atropine. If atropine fails, use pacing or a vasoactive infusion. Finally, correct all reversible triggers. Reassess after every step and call for expert help early when the patient stays unstable.
This guide covers common teaching points only. It is not a substitute for current clinical guidelines or professional medical advice.